Трампын засаг захиргаа тус улсад зөвхөн эрэгтэй, эмэгтэй гэсэн хоёр хүйсийг мөрдөх болно гэв
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Ерөнхийлөгч Д.Трамп хэлэхдээ, удахгүй Холбооны засгийн газар зөвхөн эрэгтэй, эмэгтэй гэсэн хоёр хүйсийг хүлээн зөвшөөрөх зарлигийг гаргана гэсэн.
Энэ зарлиг нь Байдены засаг захиргаанаас гаргасан хүйсийн ялгааг өргөжүүлэх, үүнд гадаад паспорт дээрх хүйсийн тэмдэглэлийг өөрчлөх оролдлогыг хүчингүй болгох зорилготой юм.
“Өнөөдрөөс эхлэн АНУ-ын засгийн газрын албан ёсны бодлого нь зөвхөн хоёр хүйс эрэгтэй, эмэгтэй байдаг гэсэн зарчимд тулгуурлана” гэж Трамп даваа гарагт тангараг өргөх ёслолын үеэр хэлж, өөрийн соёлын дайны кампанийн амлалтаа биелүүлэх эхний алхмаа хийлээ.
Трампын засаг захиргааны нэгэн албан тушаалтан даваа гарагт сэтгүүлчдэд өгсөн мэдээлэлдээ, энэ зарлиг нь “хүйсийн үзэл санааны хэт туйлшралаас эмэгтэйчүүдийг хамгаалах, биологийн бодит үнэнийг холбооны засгийн газарт сэргээхэд” чиглэгдсэн гэжээ.
Эрэгтэй, эмэгтэй гэсэн хүйс нь “өөрчлөгдөх боломжгүй бөгөөд маргаангүй бодит байдалд суурилсан” хэмээн албан тушаалтан нэмж хэлсэн байна.
Засгийн газар “хүйс” гэсэн нэр томъёог “хүйсийн ангилал” гэсэн үгээр солих бөгөөд уг хүйс нь хувь хүний “өөрчлөгдөх боломжгүй биологийн ангилал” гэж тодорхойлох аж.
Бүх төрийн байгууллагууд албан баримт бичгүүд, үүнд гадаад паспорт, виз зэрэг нь “хүйсийг зөв тусгасан” байхыг шаардана. Мөн холбооны шорон, цагаачдын хорих төвүүд, хүчирхийлэлд өртөгсдөд зориулсан байр зэрэг газруудад ганц хүйстэй орон зайг хамгаалах үүрэгтэй гэж албан тушаалтан тайлбарлажээ. Ажилчдын бүртгэл болон холбооны байгууллагуудын мэдэгдэлд энэ зарлигийг заавал мөрдөнө гэв.
2022 оноос эхлэн АНУ-ын иргэд гадаад паспорт дээрээ хүйсийн тэмдэглэгээг “X” гэж сонгох боломжтой болсон. Хүйсийн тэмдэглэгээ нь иргэний бичиг баримт эсвэл зураг бүхий үнэмлэхийн хүйстэй таарах шаардлагагүй бөгөөд хүйсийн тэмдэглэлийг өөрчлөхөд эмнэлгийн баримт бичиг шаардагдахгүй байв, үүнийг өөрчилнө гэж Төрийн департаментаас мэдээлжээ.
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## 1️⃣ What Is Trenbolone (Tren) — An Overview
| Feature | Details |
|———|———|
| **Generic name** | Trenbolone acetate or trenbolone propionate (the two most common injectable forms). |
| **Drug class** | An anabolic‑steroid, derived from nandrolone. |
| **Commercial availability** | In veterinary medicine (often as “Trenbolone Acetate” for cattle growth). Not approved for human use in the U.S., Canada, or EU. |
| **Primary uses** | • Veterinary: increase muscle mass and feed efficiency in livestock.
• Research: anabolic‑steroid studies. |
| **Key pharmacologic actions** | • Anabolic (muscle‑building).
• Androgenic (masculinizing).
• Insulin‑like effects: increased glucose uptake, glycogen synthesis. |
—
## 2. How Trenbolone Works – Mechanisms of Action
| Step | Biological Process | Effect |
|——|——————-|——–|
| **1. Cellular entry** | Trenbolone diffuses across the plasma membrane (lipophilic steroid). | It reaches the cytoplasm and nucleus. |
| **2. Receptor binding** | Binds to intracellular androgen receptors (AR) with high affinity; also binds glucocorticoid receptor (GR) to a lesser extent. | Forms Trenbolone‑AR complex. |
| **3. Nuclear translocation** | Complex enters the nucleus, attaches to specific DNA sequences called Androgen Response Elements (AREs). | Modulates transcription of target genes. |
| **4. Gene activation/repression** | Upregulation: Muscle-specific proteins (e.g., myosin heavy chain), growth factors (IGF‑1, VEGF). Downregulation: Proteolytic enzymes (atrogin‑1, MuRF1). | Leads to increased protein synthesis, reduced catabolism. |
| **5. Cellular outcomes** | Enhanced satellite cell proliferation/differentiation; angiogenesis; metabolic shifts favoring glycogen storage and lipogenesis in muscle cells. | Rapid muscle hypertrophy, improved endurance. |
### 4.2. Molecular Pathways Engaged by Testosterone
| Pathway | Key Components | Functional Consequence |
|———|—————-|————————|
| **mTORC1 signaling** | Akt → mTOR → p70S6K; S6 ribosomal protein phosphorylation | Stimulates translation initiation and elongation, increases ribosomal biogenesis. |
| **IGF‑1 autocrine/paracrine loop** | IGF‑1 secretion by muscle cells → IGF‑1R activation | Activates PI3K/Akt pathway, further promoting mTOR signaling. |
| **Transcription factor regulation** | MyoD, Myogenin upregulation; FoxO inhibition | Enhances myogenic differentiation and suppresses protein catabolism genes (atrogin‑1). |
| **Proteolytic system suppression** | Reduced ubiquitin‑proteasome activity; decreased autophagic flux | Lowers breakdown of muscle proteins. |
The net effect is a positive protein balance: synthesis exceeds degradation, leading to hypertrophy.
—
## 4. Clinical Relevance
| Context | What the evidence says |
|———|————————|
| **Athletes / Bodybuilders** | Hormonal manipulation (e.g., testosterone supplementation) can enhance muscle growth when combined with resistance training and adequate protein intake. |
| **Sarcopenia / Aging** | Low endogenous testosterone is associated with reduced muscle mass; testosterone replacement therapy has shown modest gains in lean body mass but must be weighed against cardiovascular, prostate, and erythrocytosis risks. |
| **Anabolic Steroid Abuse** | Exaggerated elevations of androgenic hormones lead to dramatic hypertrophy but carry significant adverse effects (liver toxicity, hypertension, psychiatric disturbances). |
—
## Bottom‑Line Takeaway
– **Androgen hormones (testosterone, dihydrotestosterone) directly stimulate muscle protein synthesis and reduce breakdown.**
– **They increase satellite cell activity, ribosomal biogenesis, and mitochondrial function—all key to hypertrophy.**
– **Therefore, elevated androgen levels are a major driver of increased muscle mass in men, and they can account for the larger size compared with women when other factors (nutrition, training) are similar.**
*If you’re aiming to increase muscle size, consider strategies that safely elevate endogenous testosterone—adequate sleep, resistance training, balanced nutrition—and consult a healthcare professional before using any exogenous hormones.*
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